Child Diabetes

Archive for the ‘Insulin Resistance Diabetes’ Category

Wonderlife Chromium Picolinate 200 mcg 90 Capsules Per Bottle Chromium is an essential mineral. While it may be found in a variety of foods, as many as 90 of American diets are low in chromium. Low chromium levels may result in an increase in blood sugar, triglycerides and cholesterol. Chromium supports effective functioning of insulin. Insulin is needed for transporting glucose to cells for energy production. Diabetes results when the body cannot create enough insulin or has trouble using it effectively. Chromium has been shown in numerous studies to be beneficial in treating insulin resistance and diabetes. Other studies show chromium to be effective in improving insulin sensitivity and lowering blood glucose levels. Up to 80 million Americans may suffer from insulin resistance where the body fails to respond properly to the insulin it produces. Chromium has also shown significant benefit in treating polycystic ovarian syndrome PCOS . This condition affects up to 2 million women an

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As a bodybuilder, you try to avoid sugars. Excessive sugar intake has been linked to insulin resistance, carb cravings, and bodyfat accumulation, among other things. So sucrose, or plain table sugar, is out. Fructose, while a long-lasting source of energy, has been shown in clinical studies to create gastrointestinal (GI) distress. Take too much fructose and it won’t sit well in your stomach. For optimal results, you need Carbo Plus. Carbo Plus doesn’t contain any inferior sucrose or fructose. Instead, you get glucose polymers and maltodextrin extracted from premium grain sources. Carbo Plus is unflavored and unsweetened, and each serving loads you up with 55g of pure energy. Slowly blend 3 scoops of Carbo Plus into 8 ounces of your beverage of choice. For best results use at least twice daily. One serving should be consumed within 2 hours after completion of your training or competition. Please consult your physician before starting any exercise or nutritional program. If you have diabetes, hypoglycemia or are under a physician’s care for any reason, please consult your physician before using this product. Use this product as a supplement to your normal diet, not as a sole source of nutrition. 100% Pure high performance complex carbohydrates (consisting of granulated medium length complex carbohydrates (glucose polymers) extracted from premium grain sources and maltodextrin).

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Orotates are mineral salts of orotic acid used by plants and animals to make DNA and RNA. Hans Nieper, a physician and dabbler in offbeat theories of gravitation, used orotates clinically prior to 1980. He thought that orotate salts, being neutrally charged, pass easily through cell membranes. In effect, orotate ferries the mineral atoms into cells and tissues, producing higher concentrations. Nieper promoted orotates as treatments for nearly every ailment imaginable and was consequently ignored; most of his medical claims were never rigorously tested. But in certain applications – such as athletic performance – where his ideas have recently received scientific scrutiny, they have been found to work. Nieper may have been on the right track after all. Potassium orotate is used to treat symptoms of magnesium deficiency: diabetes, insulin resistance, high blood pressure, rheumatoid arthritis, and heart disease. Nieper combined potassium and magnesium orotates to treat cardiovascular diseases. Other applications include wound healing, immune enhancement, depression and anxiety.

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I have a theory that insulin resistance of type2 diabetes is caused by man-made isomers (such as trans isomers of fatty acids) embedding into our cell membranes. Because they are geometric isomers, they can integrate into the structure of cell membranes YET still result in the loss of functionality of that component. For example binding of insulin to receptors may fail if the structure of that receptor is composed from a geometric isomer of one of it’s components.

I’m actually a physicist with a casual interest in this area. I had this idea which seemed to make sense to me . If I am correct then could we not heal ourselves of insulin resistance simply by altering our diets so that we only eat natural foods (giving up trans fats in particular) for a period of say a year will give sufficient time for the cells in our body to have been completely replaced thus healig our body of type 2 diabetes. I have heards that such measures have proved remarkably sucesfull in reducing effects

Insulin resistance is the condition in which normal amounts of insulin are inadequate to produce a normal insulin response from fat, muscle and liver cells. Insulin resistance in fat cells results in hydrolysis of stored triglycerides, which elevates free fatty acids in the blood plasma. Insulin resistance in muscle reduces glucose uptake whereas insulin resistance in liver reduces glucose storage, with both effects serving to elevate blood glucose. High plasma levels of insulin and glucose due to insulin resistance often leads to the metabolic syndrome and type 2 diabetes.

The most common type of insulin resistance is associated with obesity and metabolic syndrome. This was first described in the 1930’s by H.P. (Harry) Himsworth (University College Hospital Medical School, London). He described results of experiments in an article in 1936, entitled, "Diabetes Mellitus: Its differentiation into insulin sensitive and insulin insensitive types." He found that those with diabetes can be differentiated into two types: those in whom injected insulin produces an immediate suppression of hyperglycemia; and those in whom the insulin has little or no effect. Hyperglycemia itself can lead to insulin resistance, but N-acetylcysteine and taurine can prevent this effect[1].

Insulin resistance denotes decreased sensitivity of target cells (muscle, adipose and hepatic cells) to insulin. The very common "metabolic syndrome" is the concomitant appearance of diabetes mellitus (type 2), hypertension, combined hyperlipidemia and central obesity. It is also associated with polycystic ovarian syndrome (PCOS).

In obese patients, compensatory hyperinsulinemia reduces the expression of the membrane insulin receptor (down regulation) which maintains the maximal response. More importantly, defects in processes within the cell itself (also called post-receptor defects) appear to play a much larger role in the development of insulin resistance. A relationship between leptin resistance and insulin resistance has been suggested.

In a normal person, insulin is released from the beta cells of the Islets of Langerhans located in the pancreas after eating ("postprandial"), and it signals the body to allow glucose to enter insulin-sensitive tissues (e.g., muscle, adipose) and maintain normal blood glucose levels. In an "insulin resistant" person the message does not get through to those cells until much more insulin is released in an attempt to compensate. Occasionally, this can lead to a steep drop in blood sugar and a hypoglycaemic reaction several hours after the meal.

Insulin resistance is often associated with visceral adiposity (increased waist cicumference), hypertension, hyperglycemia and dyslipidemia involving elevated triglycerides, small dense low-density lipoprotein (sdLDL) particles, and decreased HDL cholesterol.

Insulin resistance is also often associated with a hypercoagulable state (impaired fibrinolysis) and increased inflammatory cytokine levels.

It is difficult to say that insulin resistance can be caused by isomers embedded into the cell membranes (fatty acids) only since Insulin resistance is also associated in hypercoagulable states. Also, there are other possible causes of Insulin resistance such as prolonged use of medications like Rifampicin, Isoniazid, Olanzapine, Risperidone, Antiretrovirals. Lastly, genetic causes have been addressed such as the Donohue Syndrome which displays insulin receptor mutations, and LMNA mutations (Familial Partial Lipodystrophy).

I have a theory that insulin resistance of type2 diabetes is caused by man-made isomers (such as trans isomers of fatty acids) embedding into our cell membranes. Because they are geometric isomers, they can integrate into the structure of cell membranes YET still result in the loss of functionality of that component. For example binding of insulin to receptors may fail if the structure of that receptor is composed from a geometric isomer of one of it’s components.

I’m actually a physicist with a casual interest in this area. I had this idea which seemed to make sense to me . If I am correct then could we not heal ourselves of insulin resistance simply by altering our diets so that we only eat natural foods (giving up trans fats in particular) for a period of say a year will give sufficient time for the cells in our body to have been completely replaced thus healig our body of type 2 diabetes. I have heards that such measures have proved remarkably sucesfull in reducing effects

Insulin resistance is the condition in which normal amounts of insulin are inadequate to produce a normal insulin response from fat, muscle and liver cells. Insulin resistance in fat cells results in hydrolysis of stored triglycerides, which elevates free fatty acids in the blood plasma. Insulin resistance in muscle reduces glucose uptake whereas insulin resistance in liver reduces glucose storage, with both effects serving to elevate blood glucose. High plasma levels of insulin and glucose due to insulin resistance often leads to the metabolic syndrome and type 2 diabetes.

The most common type of insulin resistance is associated with obesity and metabolic syndrome. This was first described in the 1930’s by H.P. (Harry) Himsworth (University College Hospital Medical School, London). He described results of experiments in an article in 1936, entitled, "Diabetes Mellitus: Its differentiation into insulin sensitive and insulin insensitive types." He found that those with diabetes can be differentiated into two types: those in whom injected insulin produces an immediate suppression of hyperglycemia; and those in whom the insulin has little or no effect. Hyperglycemia itself can lead to insulin resistance, but N-acetylcysteine and taurine can prevent this effect[1].

Insulin resistance denotes decreased sensitivity of target cells (muscle, adipose and hepatic cells) to insulin. The very common "metabolic syndrome" is the concomitant appearance of diabetes mellitus (type 2), hypertension, combined hyperlipidemia and central obesity. It is also associated with polycystic ovarian syndrome (PCOS).

In obese patients, compensatory hyperinsulinemia reduces the expression of the membrane insulin receptor (down regulation) which maintains the maximal response. More importantly, defects in processes within the cell itself (also called post-receptor defects) appear to play a much larger role in the development of insulin resistance. A relationship between leptin resistance and insulin resistance has been suggested.

In a normal person, insulin is released from the beta cells of the Islets of Langerhans located in the pancreas after eating ("postprandial"), and it signals the body to allow glucose to enter insulin-sensitive tissues (e.g., muscle, adipose) and maintain normal blood glucose levels. In an "insulin resistant" person the message does not get through to those cells until much more insulin is released in an attempt to compensate. Occasionally, this can lead to a steep drop in blood sugar and a hypoglycaemic reaction several hours after the meal.

Insulin resistance is often associated with visceral adiposity (increased waist cicumference), hypertension, hyperglycemia and dyslipidemia involving elevated triglycerides, small dense low-density lipoprotein (sdLDL) particles, and decreased HDL cholesterol.

Insulin resistance is also often associated with a hypercoagulable state (impaired fibrinolysis) and increased inflammatory cytokine levels.

It is difficult to say that insulin resistance can be caused by isomers embedded into the cell membranes (fatty acids) only since Insulin resistance is also associated in hypercoagulable states. Also, there are other possible causes of Insulin resistance such as prolonged use of medications like Rifampicin, Isoniazid, Olanzapine, Risperidone, Antiretrovirals. Lastly, genetic causes have been addressed such as the Donohue Syndrome which displays insulin receptor mutations, and LMNA mutations (Familial Partial Lipodystrophy).

No not really, diabetes is formed when your pancreas stop producing insullin.

No not really, diabetes is formed when your pancreas stop producing insullin.

Insulin plays an important role in weight management both in controlling body fat and building muscle tissue Insulin resistance has been linked to obesity heart disease diabetes and hypertension Chromium Picolinate aids the bodys metabolism by helping insulin function efficiently Each capsule contains 200mcg of biologically-active and yeast-free chromium picolinateIt is the perfect formula for those looking to supplement with a smaller dose of chromium For a larger amount of chromium Universal offers Chromax II

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Insulin plays an important role in weight management both in controlling body fat and building muscle tissue Insulin resistance has been linked to obesity heart disease diabetes and hypertension Chromium Picolinate aids the bodys metabolism by helping insulin function efficiently Each capsule contains 200mcg of biologically-active and yeast-free chromium picolinateIt is the perfect formula for those looking to supplement with a smaller dose of chromium For a larger amount of chromium Universal offers Chromax II

Read the rest of this entry »

By lunch, my blood sugar (after having a good breakfast) it is between 80 and 90mg/dL. Do I need to start medication like glucaphage?

Cut down on carbs. And get to exersising at least walk 1/2 hour a day. After a couple of weeks check your glucose levels and see.

There are various methods to determine blood glucose level. Some tests give you accurate diagnosis of diabetes or pre-diabetes, while others will tell you how well you are managing your diabetes.

Fasting Blood Sugar Test:
Measures the blood sugar level after 8 hours fast or overnight. Normal fasting blood glucose level is less than 100mg/dl. If your fasting blood glucose level is from 100mg/dl to 125mg/dl then you will have impaired blood glucose level also known as Pre-Diabetes. If your blood glucose level is above 125mg/dl then your doctor will diagnose as a patient of diabetes. To confirm the diagnosis, your doctor may repeat the fasting blood glucose test on any other day. If you have blood glucose level of 126mg/dl or higher in two consecutive tests, then you may have diabetes. If you have blood glucose level greater than 200mg/dl and you have symptoms of diabetes like increased thirst or hunger, frequent urination, weight loss, blurred vision etc, then you may be diagnosed with diabetes mellitus without confirming it with second test.

Random Blood Glucose Test:
Random blood Glucose test gives your blood sugar at any time in a day. Normal random blood sugar level should be less than 200mg/dl. If your random blood glucose level is between 140mg/dl to 200mg/dl then you will have pre-diabetes.

Oral glucose tolerance test
This test measures your response to sugar. First we measure fasting blood glucose level, and then glucose solution is given, after that we measure blood glucose after 1 hour and 2hours. A normal blood glucose level after an oral glucose tolerance test is less than 140 mg/dL. Level between 140 mg/dL to 199 mg/dL suggests pre-diabetes. A blood glucose level of 200 mg/dL or higher two hours after you drink the glucose solution may suggest that you have diabetes mellitus.

Glycated hemoglobin (A1C) test
This test is not for diagnosing diabetes, but it shows you how well you have controlled your sugar in last 2 or 3 months. Normal value is less than 7%, however if it is more than 7 then you and your doctor should think of changing your treatment of diabetes.

Always Remember, your blood glucose measurement alone is not enough to differentiate between type 1 and type 2 diabetes. Your doctor may do some other tests to find out which type of diabetes you have.
Good luck my friend